Palomar has developed the RejuveLux™ Photofacial process for the treatment of pigmented and vascular lesions on the face, as well as the arms, chest, and other large areas. The RejuveLux process is able to clear sunspots, rosacea, telangiectasias, and other conditions, in order to improve skin tone and texture.
Treating a pigmented lesion with the pulsed light results in heating the melanin. The damaged lesion is then naturally sloughed as the skin exfoliates. Similar results are seen in the treatment of vascular lesions. The absorption of the light in oxyhemoglobin, de-oxyhemoglobin, and the resultant thermal action inside the blood vessels result in intraluminal coagulation, vasoconstriction, and damage to the vessel endothelial lining, all of which contribute to the degeneration and disappearance of any visible vessels.
Currently, there are two accepted non-ablative methods of skin rejuvenation:
one that goes after microvasculature in the papillary plexus and one that utilizes
bulk tissue heating through water absorption. Because the RejuveLux process
delivers light that is well absorbed by oxyhemoglobin and deoxyhemoglobin in
the papillary dermis, as well as infrared light absorbed by water, it combines
the effects of both skin rejuvenation methods. The microvasculature of the dermis
is heated without creating side effects and with minimal patient discomfort
or downtime.
This technique involves the deposition of heat into the vasculature and tissue
of the upper dermis while minimizing the heating of the epidermis by cooling
the surface of the skin with the handpiece’s cryogen-cooled sapphire lens.
The heat-stimulated dermis releases mediators and initiates fibroblast activity,
which induces the formation of collagen. Increased collagen production ultimately
reduces fine lines, fills in very mild wrinkles in the face and neck, and smoothes
the skin, reducing pore size and improving skin texture.
The mechanism of pigmented lesion removal is based upon selective destruction of melanocytes and melanosome-containing epidermal keratinocytes. The lesion darkens and crusts after treatment, and is naturally sloughed off the skin within two weeks.
The absorption of pulsed light in oxyhemoglobin, de-oxyhemoglobin, and the resultant thermal action inside the blood vessels results in intraluminal coagulation, vasoconstriction, and damage to the vessel endothelial lining, all of which contribute to the degeneration and disappearance of any visible vessels